vasa vasorum การใช้

• Vasa vasorum are more frequent in veins than arteries.
• This would lead to more vasa vasorum needed to supply the vessels sufficiently.
• The structure of the vasa vasorum varies with the size, function and location of the vessels.
• It typically causes inflammation of the network of small vessels ( vasa vasorum ) that supplies the larger arteries.
• The second hypothesis suggests that the vasa vasorum ruptures and causes a hemorrhage in the wall of the aorta.
• In the smallest vessels, the vessels'own circulation nourishes the walls directly and they have no vasa vasorum at all.
• The third stage of syphilis also manifests as aneurysm of the aorta, which is due to loss of the vasa vasorum in the tunica adventitia.
• Some authorities hypothesize that the vasa vasorum would be more abundant in large veins, as partial oxygen pressure and osmotic pressure is lower in veins.
• Interestingly, T-cadherin is highly expressed in adventitial vasa vasorum of injured arteries suggesting the involvement of " -cadherin in the processes of angiogenesis after vessel injury.
• Depending on the type of vasa vasorum, it penetrates the vessel wall starting at the intimal layer ( vasa vasorum interna ) or the adventitial layer ( vasa vasorum externa ).
• Depending on the type of vasa vasorum, it penetrates the vessel wall starting at the intimal layer ( vasa vasorum interna ) or the adventitial layer ( vasa vasorum externa ).
• Depending on the type of vasa vasorum, it penetrates the vessel wall starting at the intimal layer ( vasa vasorum interna ) or the adventitial layer ( vasa vasorum externa ).
• In the largest vessels, the vasa vasorum penetrates the outer ( tunica adventitia ) layer and middle ( tunica media ) layer almost to the inner ( tunica intima ) layer.
• This means that it would take longer for any oxygen to diffuse through to the cells in the tunica adventitia and the tunica media, causing them to need a more extensive vasa vasorum.
• Due to higher radial and circumferential pressures within the vessel wall layers closer to the main lumen of the artery, vasa vasorum externa cannot perfuse these regions of the vessel wall ( occlusive pressure ).
• It has been noted that the MYH11 mutation could be responsible for at least 14 % of hereditary aortic media has been recorded in these individuals, with areas of disorganization and hyperplasia as well as stenosis of the aorta s vasa vasorum.
• Instead, the platelet-derived growth factor degranulated by the clotted platelets will attract a layer of smooth muscle cells to cover the clot, and this layer of mural smooth muscle will be vascularised by the blood inside the vessel lumen rather than by the vasa vasorum.
• This is most likely due to a tear in the tunica intima ( the inner layer ), allowing blood to enter the tunica media, although other lines of evidence have suggested that the blood may instead arise from the vasa vasorum, the small blood vessels that supply the outer layer of larger blood vessels.